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There are many chemicals in the
brain that communicate messages within it and in the nervous system throughout
the body. These are called neurons that interact through chemical messengers
called neurotransmitter and they communicated to all the neurons that
constantly communicating together while interchanging neurotransmitter.

The axon (threadlike structure) is the
body of the neurons. There are small spaces between the neurons called synapse.
This is how the neurons send and receive messages. The received neuron triggers
whatever chemical it received and transmit it down to other neurons.

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The receiving end of the neurons
has receptors, once the chemical signal is received through the synapse the
receptors are triggered and the neurotransmitter send the message to the next
neuron. 

            Neurotransmitters and synapses need to reset
between messages to be able to receive the next neurotransmitter and deactivate
causing the receptors
to relax and release the next neurotransmitter back into the synapse where they
are taken back up by the original neuron which sent the message (this is the
reuptake process). The neurotransmitter and reparable and reused for the
message to reach the synapse.

            The brain do this very quickly throughout the entire
cycle and when this cycle is disrupted or any problems occurs it could cause a
dysfunction of the chemical flow in the brain and nervous system.

Depression can cause a connection
problem or variation in the brain, particularly involving the neurotransmitter
serotonin, norepinephrine, and dopamine.

The extent of neurotransmitters in
the brain is not measurable nor are the activity of them

There are antidepressants medication to treat indication of depression.
The antidepressant can alter the neurotransmitters and their receptors.

Depression is a persistent mental
disorder causing one to feel sorrow, to be short temper, with irregularities in
appetite and sleep. The biogenic amine hypothesis suggests that the disorder is
related with a flaw in the serotonin (5-hydroxytryptamine, 5-HT), noradrenaline
(NA), and acetylcholine (ACh) which are these neurotransmitter (Wang, el at., 2016).

Evidence has shown that change in
the brain serotonergic circuitries are entangle in the functional change of
depression and that 5-HT receptor dysfunction could play a crucially part to
the occurrence of depression (Wang,
el at., 2016).

Serotonin
is generated by serotonergic neurons which controls bodily functions, moods,
etc., and if the produced in serotonin decreases by these neurons can cause
depression, with some people mood state changing and causing them to feel
suicidal. 

Early
explanation of (noradrenaline) aromatic amines (NA) that includes
neurotransmitter such as epinephrine and dopamine, suggests that certain areas
of the brain is to blame for creating depression feelings, showing low levels
of noradrenaline.

Recent
studies have also shown that people who have never shown signs of depression
than people with more than one incident of depressive disorder had fewer
norepinephrinergic neurotransmitter, but this is not necessarily true in all
people. Some people show normal signs in the production of norepinephrine neurons,
while low levels of serotonin provoke a drop in norepinephrine leading to
depression. Some people are impotent to depression and their norepinephrine
system cannot recognize or respond to stress as much as others.

Dopamine neurotransmitter connected
to depression, enhances rewards and ability to feel pleasure. Low dopamine may
account for why people who are depressed do not feel the same feelings of
pleasure even before they get depressed.

Dopamine neurons gives us the
feeling of rewards and motivation. There is medication treatment of
antidepressant and brain activation therapies that can impact the complicated dopamine
structure (Tye, et al. 2016).

 Studies have shown that inhibition or
excitation of specified midbrain dopamine neurotransmitters instantly, and
allowing movement in two different directions (induces or relieves) various
unconstrained symptoms caused by persistent tension (Tye, et al. 2016).

Using light to control genetic
cells in the neurons alters the nervous system encryption of behaviors related
to depression in the downstream of hypothalamus (basal forebrain, to the
preoptic area) with rodents moving freely, suggesting this processes affect
depression symptoms may need alteration in the nervous system (Tye, et al. 2016).

Acetylcholine (ACh) is a biological chemical
that functions as a neurotransmitter in the brain and body that
releases chemicals by nerve cells to send signals to other cells. The chemical is
compound of acetic acid and choline. ACh neurotransmitters activate the nervous
structure in the muscle and autonomic nervous system (ANS), smoothing the muscles and glands, and
controlling the heartbeat, respiratory tracts, digestion and sexual stimulation (Schober, et al.).

Acetylcholine operates as
neurotransmitter and neuromodulator. There are many cholinergic areas with all
of them having separated functionally roles like rousing, awareness,
recollection, and drive (Schober, et al.).

 

The postsynaptic neuron supports
the receptors for the neurotransmitter that is released in the synaptic cleft
by the presynaptic neuron preganglionic fiber in the autonomic nervous
structure fibers from the CNS to the preganglionic fabrics. All the preganglionic
neurotransmitters are in the in the sympathetic and the parasympathetic
section of the ANS. There are two classes of cholinergic receptors (nicotinic
and muscarinic). Nicotinic (N1) receptors are on the
body of the postganglionic and speed up by sending impulses down to the axon
releasing the neurotransmitter. Found only in the chromaffin cells of the
adrenal medulla increases the quantity of epinephrine and norepinephrine
produced in the blood (Massey, et
al. 2001).

Muscarinic receptors act different than the N1 receptor. They
form G-proteins (phosphorylate) in the cell membranes of certain neurons and
other cells.

There are drugs targeting nicotine
acetylcholine receptors to treat depression. Cholinergic neurotransmission
modulate dopamine functions.

Depression is caused by imbalance
of brain chemicals and a persistent mental disorder causing one to feel sorrow,
to be short temper, with irregularities in appetite and sleep. Depression can
cause a connection problem or variation in the brain, particularly involving
the neurotransmitter serotonin, norepinephrine, and dopamine.

Selective serotonin reuptake inhibitors (SSRIs), antidepressants, are
drugs for treatment of depression altering the neurotransmitters and
their receptors.

 

There are many chemicals in the
brain that communicate messages within it and in the nervous system throughout
the body. These are called neurons that interact through chemical messengers
called neurotransmitter and they communicated to all the neurons that
constantly communicating together while interchanging neurotransmitter.

The axon (threadlike structure) is the
body of the neurons. There are small spaces between the neurons called synapse.
This is how the neurons send and receive messages. The received neuron triggers
whatever chemical it received and transmit it down to other neurons.

The receiving end of the neurons
has receptors, once the chemical signal is received through the synapse the
receptors are triggered and the neurotransmitter send the message to the next
neuron. 

            Neurotransmitters and synapses need to reset
between messages to be able to receive the next neurotransmitter and deactivate
causing the receptors
to relax and release the next neurotransmitter back into the synapse where they
are taken back up by the original neuron which sent the message (this is the
reuptake process). The neurotransmitter and reparable and reused for the
message to reach the synapse.

            The brain do this very quickly throughout the entire
cycle and when this cycle is disrupted or any problems occurs it could cause a
dysfunction of the chemical flow in the brain and nervous system.

Depression can cause a connection
problem or variation in the brain, particularly involving the neurotransmitter
serotonin, norepinephrine, and dopamine.

The extent of neurotransmitters in
the brain is not measurable nor are the activity of them

There are antidepressants medication to treat indication of depression.
The antidepressant can alter the neurotransmitters and their receptors.

Depression is a persistent mental
disorder causing one to feel sorrow, to be short temper, with irregularities in
appetite and sleep. The biogenic amine hypothesis suggests that the disorder is
related with a flaw in the serotonin (5-hydroxytryptamine, 5-HT), noradrenaline
(NA), and acetylcholine (ACh) which are these neurotransmitter (Wang, el at., 2016).

Evidence has shown that change in
the brain serotonergic circuitries are entangle in the functional change of
depression and that 5-HT receptor dysfunction could play a crucially part to
the occurrence of depression (Wang,
el at., 2016).

Serotonin
is generated by serotonergic neurons which controls bodily functions, moods,
etc., and if the produced in serotonin decreases by these neurons can cause
depression, with some people mood state changing and causing them to feel
suicidal. 

Early
explanation of (noradrenaline) aromatic amines (NA) that includes
neurotransmitter such as epinephrine and dopamine, suggests that certain areas
of the brain is to blame for creating depression feelings, showing low levels
of noradrenaline.

Recent
studies have also shown that people who have never shown signs of depression
than people with more than one incident of depressive disorder had fewer
norepinephrinergic neurotransmitter, but this is not necessarily true in all
people. Some people show normal signs in the production of norepinephrine neurons,
while low levels of serotonin provoke a drop in norepinephrine leading to
depression. Some people are impotent to depression and their norepinephrine
system cannot recognize or respond to stress as much as others.

Dopamine neurotransmitter connected
to depression, enhances rewards and ability to feel pleasure. Low dopamine may
account for why people who are depressed do not feel the same feelings of
pleasure even before they get depressed.

Dopamine neurons gives us the
feeling of rewards and motivation. There is medication treatment of
antidepressant and brain activation therapies that can impact the complicated dopamine
structure (Tye, et al. 2016).

 Studies have shown that inhibition or
excitation of specified midbrain dopamine neurotransmitters instantly, and
allowing movement in two different directions (induces or relieves) various
unconstrained symptoms caused by persistent tension (Tye, et al. 2016).

Using light to control genetic
cells in the neurons alters the nervous system encryption of behaviors related
to depression in the downstream of hypothalamus (basal forebrain, to the
preoptic area) with rodents moving freely, suggesting this processes affect
depression symptoms may need alteration in the nervous system (Tye, et al. 2016).

Acetylcholine (ACh) is a biological chemical
that functions as a neurotransmitter in the brain and body that
releases chemicals by nerve cells to send signals to other cells. The chemical is
compound of acetic acid and choline. ACh neurotransmitters activate the nervous
structure in the muscle and autonomic nervous system (ANS), smoothing the muscles and glands, and
controlling the heartbeat, respiratory tracts, digestion and sexual stimulation (Schober, et al.).

Acetylcholine operates as
neurotransmitter and neuromodulator. There are many cholinergic areas with all
of them having separated functionally roles like rousing, awareness,
recollection, and drive (Schober, et al.).

 

The postsynaptic neuron supports
the receptors for the neurotransmitter that is released in the synaptic cleft
by the presynaptic neuron preganglionic fiber in the autonomic nervous
structure fibers from the CNS to the preganglionic fabrics. All the preganglionic
neurotransmitters are in the in the sympathetic and the parasympathetic
section of the ANS. There are two classes of cholinergic receptors (nicotinic
and muscarinic). Nicotinic (N1) receptors are on the
body of the postganglionic and speed up by sending impulses down to the axon
releasing the neurotransmitter. Found only in the chromaffin cells of the
adrenal medulla increases the quantity of epinephrine and norepinephrine
produced in the blood (Massey, et
al. 2001).

Muscarinic receptors act different than the N1 receptor. They
form G-proteins (phosphorylate) in the cell membranes of certain neurons and
other cells.

There are drugs targeting nicotine
acetylcholine receptors to treat depression. Cholinergic neurotransmission
modulate dopamine functions.

Depression is caused by imbalance
of brain chemicals and a persistent mental disorder causing one to feel sorrow,
to be short temper, with irregularities in appetite and sleep. Depression can
cause a connection problem or variation in the brain, particularly involving
the neurotransmitter serotonin, norepinephrine, and dopamine.

Selective serotonin reuptake inhibitors (SSRIs), antidepressants, are
drugs for treatment of depression altering the neurotransmitters and
their receptors.

 

 

 

  

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